Search results for "PsyArXiv|Neuroscience|Behavioral Neuroscience"

showing 10 items of 8055 documents

Clinical significance of the quantitative and qualitative determination of the cerebral blood flow through the use of isotopes

1966

Es wird über die bisher entwickelten Methoden zur bestimmung der Hirndurchblutung mit radioaktiven Isotopen berichtet, wobei die quantitativen Untersuchungsmethoden hinsichtlich ihrer klinischen Bedeutung den qualitativen gegenübergestellt werden. Während die quantitativen Methoden eine genaue Bestimmung des cerebralen Blutvolumens und damit der Gesamtdurchblutung des Hirns bzw. bestimmter Hirnareale ermöglichen, bieten die qualitativen Untersuchungsmethoden hinsichtlich der klinisch-diagnostischen Aussagekraft besondere Vorteile. Aus der Bestimmung der Zirkulationszeit des Hirns und dem Verlauf der über beiden Hemisphären registrierten Aktivitätskurven lassen sich differenzial-diagnostisch…

medicine.medical_specialtymedicine.diagnostic_testbusiness.industryArteriovenous malformationmedicine.diseaseEntire brainSurgerylcsh:RC321-571NeurologyCerebral blood flowOlder patientsCirculatory timeAngiographymedicineClinical significanceNeurology (clinical)Radiologybusinesslcsh:Neurosciences. Biological psychiatry. NeuropsychiatryArquivos de Neuro-Psiquiatria
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Sleep restriction for the duration of a work week impairs multitasking performance

2010

It is important to develop shift schedules that minimise the chance for sleep-related human error in safety-critical domains. Experimental data on the effects of sleep restriction (SR) play a key role in this development work. In order to provide such data, we conducted an experiment in which cognitively demanding and long-duration task performance, simulating task performance at work, was measured under SR and following recovery. Twenty healthy male volunteers, aged 19-29 years, participated in the study. Thirteen of them had first two baseline days (8-h sleep opportunity per day), then five SR days (4-h sleep) and finally two recovery days (8-h sleep). Seven controls were allowed to sleep…

medicine.medical_specialtymedicine.diagnostic_testbusiness.industryCognitive Neuroscience05 social sciencesPsychomotor vigilance taskPoison controlGeneral MedicineElectrooculographyAudiologyElectroencephalographySleep in non-human animals050105 experimental psychology03 medical and health sciencesBehavioral Neuroscience0302 clinical medicineRhythmmedicineHuman multitasking0501 psychology and cognitive sciencesbusiness030217 neurology & neurosurgerySleep restrictionJournal of Sleep Research
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Choroidal filling pattern during a hypotensive crisis

1987

Observation d'une jeune femme de 18 ans atteinte de dystrophie progressive des cones et bâtonnets et ayant presente une crise d'hypotension arterielle au cours d'une angiographie fluoresceinique

medicine.medical_specialtymedicine.diagnostic_testbusiness.industryEye diseaseArterial hypotensionmedicine.diseaseSensory SystemsSurgeryCellular and Molecular NeuroscienceOphthalmologymedicine.anatomical_structureOphthalmologyAngiographymedicineChoroidbusinessRetinopathyGraefes Archive for Clinical and Experimental Ophthalmology
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Early developmental alterations of low-Mg2+ -induced epileptiform activity in the intact corticohippocampal formation of the newborn mouse in vitro.

2005

Abstract The generation, propagation and pharmacological properties of low-Mg 2+ -induced epileptiform activity were examined in the intact corticohippocampal formation (CHF) of the newborn (P0–4) mouse in vitro. Multi-site field potential recordings in dentate gyrus (DG), CA3, CA1, entorhinal cortex (EC) and temporal cortex (TC) revealed in 0.2 mM Mg 2+ -containing ACSF a stable pattern of spontaneous epileptiform activity consisting of recurrent ictal-like events (ILEs) and interictal events (IEs). Although this activity could be consistently observed as early as P0, ILEs were smaller in amplitude, less frequent and showed a slower onset in P0–2 as compared to P3–4 animals. In all age gro…

medicine.medical_specialtymedicine.drug_classHippocampusAction PotentialsKainate receptorAMPA receptorBiologyHippocampusStatistics NonparametricMiceOrgan Culture TechniquesInternal medicineNeural PathwaysmedicineLimbic SystemAnimalsMagnesiumMolecular BiologyTemporal cortexCerebral CortexEpilepsyGeneral NeuroscienceDentate gyrusAntagonistAge FactorsReceptor antagonistEntorhinal cortexElectrophysiologyMice Inbred C57BLDisease Models AnimalEndocrinologynervous systemAnimals NewbornNeurology (clinical)NeuroscienceMagnesium DeficiencyDevelopmental BiologyBrain research
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From structural biochemistry to expression profiling: Neuroprotective activities of estrogen

2005

Abstract Estrogens are neuromodulatory and neuroprotective hormones. Chemically, estrogens are steroid compounds and unfold most of their activities through the activation of nuclear receptors that bind to specific target genes and control their transcription. Two subtypes of estrogen receptors are known (estrogen receptor α and estrogen receptor β) and they are expressed throughout the body including the CNS and in particular the brain. We employed large scale DNA-chip-analysis to display the gene expression pattern differentially regulated by both estrogen receptor subtypes in human neuronal cells. We identified different gene families regulated by estrogen receptors that complement the k…

medicine.medical_specialtymedicine.drug_classModels NeurologicalEstrogen receptorBiologyNeuroprotectionAntioxidantsCell Line TumorInternal medicinemedicineHumansEstrogen receptor betaPELP-1EstradiolGene Expression ProfilingGeneral NeuroscienceBrainEstrogensCell biologyGene expression profilingNeuroprotective AgentsEndocrinologyReceptors EstrogenNuclear receptorEstrogenFemaleNervous System Diseaseshormones hormone substitutes and hormone antagonistsHormoneNeuroscience
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Ovarian and Adrenal Hyperandrogenism

2007

Because in normal women androgens are secreted in almost equal quantities by both adrenals and ovaries, for many years many studies have tried to distinguish the source of androgen excess. However, in the last 10-15 years, the diagnoses of ovarian or adrenal hyperandrogenism have almost disappeared. This is due to the lack of specificity of dynamic tests as well as to the emphasis given on clinical information and ovarian sonography for the diagnosis of hyperandrogenic syndromes. However, determination of the source of increased androgens may still be useful for improving the classification and the understanding of androgen excess disorders. The aim of this review is to examine the source o…

medicine.medical_specialtymedicine.drug_classidiopathic hyperandrogenismOvaryurologic and male genital diseasesAndrogen ExcessGeneral Biochemistry Genetics and Molecular Biologychemistry.chemical_compoundDehydroepiandrosterone sulfateNCAHHistory and Philosophy of ScienceInternal medicineAdrenal GlandsPCOSmedicineHyperinsulinemiaHumansbusiness.industryGeneral NeuroscienceOvaryHyperandrogenismnonclassic 21-hydroxylase deficiencymedicine.diseaseAndrogenPolycystic ovaryAndrogen secretionEndocrinologymedicine.anatomical_structurechemistryFemaleSteroid 21-Hydroxylaseandrogen excessHyperandrogenismbusinessPolycystic Ovary SyndromeAnnals of the New York Academy of Sciences
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Activation of mGlu3 Receptors Stimulates the Production of GDNF in Striatal Neurons

2009

Metabotropic glutamate (mGlu) receptors have been considered potential targets for the therapy of experimental parkinsonism. One hypothetical advantage associated with the use of mGlu receptor ligands is the lack of the adverse effects typically induced by ionotropic glutamate receptor antagonists, such as sedation, ataxia, and severe learning impairment. Low doses of the mGlu2/3 metabotropic glutamate receptor agonist, LY379268 (0.25-3 mg/kg, i.p.) increased glial cell line-derived neurotrophic factor (GDNF) mRNA and protein levels in the mouse brain, as assessed by in situ hybridization, real-time PCR, immunoblotting, and immunohistochemistry. This increase was prominent in the striatum, …

medicine.medical_specialtymedicine.drug_classlcsh:MedicineSubstantia nigraReceptors Metabotropic GlutamateSettore BIO/09 - FisiologiaPolymerase Chain ReactionMiceNeurotrophic factorsInternal medicinemedicineGlial cell line-derived neurotrophic factorAnimalsGlial Cell Line-Derived Neurotrophic FactorRNA MessengerAmino Acidslcsh:ScienceReceptorIn Situ HybridizationNeurological Disorders/Movement DisordersNeuronsMultidisciplinarybiologyNeuroscience/Neuronal and Glial Cell Biologylcsh:RGlutamate receptorBridged Bicyclo Compounds HeterocyclicReceptor antagonistCorpus StriatumEndocrinologyMetabotropic receptornervous systemMetabotropic glutamate receptorSettore BIO/14 - Farmacologiabiology.proteinlcsh:QNeuroscience/Neurobiology of Disease and RegenerationReceptors Metabotropic Glutamate/agonists Glial Cell Line-Derived Neurotrophic FactorResearch Article
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Possible Pathogenetic Relevance of Interleukin-1beta in "Destructive" Organ-specific Autoimmune Disease (Hashimoto's Thyroiditis)

1999

Thyroid follicular cells (TFC) abundantly express a variety of immunologically relevant surface molecules in Hashimoto's thyroiditis (HT), for example, MHC antigens and adhesion molecules such as ICAM-1. Cytokines produced by infiltrating type 1 helper and cytotoxic T cells are importantly involved in de novo expression or up-regulation of such molecules. We recently demonstrated that TFC from HT patients almost invariably bear on their surface two additive functional molecules: Fas/Apo1/CD95, an important participant in apoptosis, and B7.1, a member of a family of "co-stimulatory" molecules that are crucial for efficient antigen presentation. To date, 12 out of 14 surgical HT thyroid speci…

medicine.medical_specialtymedicine.medical_treatmentAntigen presentationThyroid Glandmedicine.disease_causeGeneral Biochemistry Genetics and Molecular BiologyFas ligandAutoimmunityHistory and Philosophy of ScienceInternal medicinemedicineHumansCytotoxic T cellfas ReceptorChemistryGeneral NeuroscienceThyroiditis AutoimmuneInterleukinFas receptorMolecular biologyGraves DiseaseRecombinant ProteinsCytokineEndocrinologyApoptosisB7-1 AntigenCytokinesInterleukin-1Annals of the New York Academy of Sciences
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Hyperinsulinemia, hyperproinsulinemia and insulin resistance in the metabolic syndrome.

1996

For better comprehension of the metabolic syndrome, it is necessary to differentiate the effect of insulin on glucose metabolism on the one hand, and on other metabolic activities on the other hand. Whereas glucose utilization is affected by insulin resistance, the effect of insulin on lipid metabolism, ion and aminoacid transport does not seem to be diminished. Lipid metabolism, however, seems to play a crucial role in the induction of the vicious cycle. Increased energy and fat ingestion may be due to an increased number of galanin secreting cells in the hypothalamus. The excessive fat intake results in an increased rate of release of insulin and increased influx of triglycerides into the…

medicine.medical_specialtymedicine.medical_treatmentCarbohydrate metabolismFatty Acids NonesterifiedHyperproinsulinemiaModels BiologicalCellular and Molecular NeuroscienceInsulin resistanceInternal medicineHyperinsulinismmedicineHyperinsulinemiaAnimalsHomeostasisHumansInsulinMolecular BiologyTriglyceridesPharmacologyChemistryInsulinCell BiologySyndromemedicine.diseaseEndocrinologyAminoacid transportMolecular MedicineMetabolic syndromeInsulin ResistanceHyperinsulinismProinsulinExperientia
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Cardiac pacemaker function of HCN4 channels in mice is confined to embryonic development and requires cyclic AMP.

2008

Important targets for cAMP signalling in the heart are hyperpolarization-activated and cyclic nucleotide-gated (HCN) channels that underlie the depolarizing 'pacemaker' current, I(f). We studied the role of I(f) in mice, in which binding of cAMP to HCN4 channels was abolished by a single amino-acid exchange (R669Q). Homozygous HCN4(R669Q/R669Q) mice die during embryonic development. Prior to E12, homozygous and heterozygous embryos display reduced heart rates and show no or attenuated responses to catecholaminergic stimulation. Adult heterozygous mice display normal heart rates at rest and during exercise. However, following beta-adrenergic stimulation, hearts exhibit pauses and sino-atrial…

medicine.medical_specialtymedicine.medical_treatmentCyclic Nucleotide-Gated Cation ChannelsEmbryonic DevelopmentStimulationMice TransgenicBiologyIn Vitro TechniquesGeneral Biochemistry Genetics and Molecular BiologyCardiac pacemakerArticleMiceHeart RatePregnancyInternal medicineHeart ratemedicineCyclic AMPHyperpolarization-Activated Cyclic Nucleotide-Gated ChannelsAnimalsMyocytes CardiacMolecular BiologyIon channelCells CulturedCatecholaminergicGeneral Immunology and MicrobiologyGeneral NeuroscienceEmbryogenesisDepolarizationEmbryoHeartMice Inbred C57BLEndocrinologyMutationFemaleThe EMBO journal
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